Hi Steven,

Thanks for taking the time to respond to the article! To address the 3 comments you left:

#1 – My article is based on a straw man argument & I failed to mention insulin resistance

For reference, a straw man is an informal fallacy based on giving the impression of refuting an opponent’s argument, while actually refuting an argument that was not presented by that opponent. Throughout my article, I was refuting the stance that insulin causes weight gain. Perhaps you interpreted it in a different way? And if so, would you care to elaborate on just how, exactly, my article has distorted the opposing argument?

It’s quite interesting for you to have brought up insulin resistance. If I’ve understood you correctly, you believe that insulin resistance leads to (i.e. causes) obesity, Type 2 diabetes, and more. Now–insulin resistance is indeed a precursor to type 2 diabetes. But here’s a question to think about: what causes insulin resistance in the first place? Even researchers aren’t entirely sure yet. What has been established is that people who are obese (particularly those with higher amounts of visceral fat) are more likely to be insulin resistant. BUT losing excess body fat often resolves insulin resistance. It thus seems to suggest that excess body fat is the precursor to insulin resistance, instead of the other way round.

#2 – That low-carb diet studies aren’t really low carb

There have been multiple studies that are indeed low carb (<5% or <50 g of carbs). There still doesn’t appear to be superior weight loss results compared to other diets or calorie restriction.







#3 – Protein does not stimulate insulin production when in ketosis

This is the first time I’ve read of this–where did you get the information from?

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